Lately, my skin feels…

Red and flushed, most of the time: why midlife skin stays red

A hot flush is only part of it. Midlife redness usually comes from three things stacked: a thinner barrier, vessels that stay reactive, and in some women rosacea.

Cōpḍī Prep bottle with dried nigella flowers and prickly pear

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If your face has started holding colour, high in the cheeks, across the nose, sometimes the chin, and it no longer fades the way it used to, you are not imagining a change. Three things tend to be happening at once in midlife skin, and a hot flush is only one of them.

1. A hot flush is blood vessels opening, not inflammation

A hot flush starts in the brain, not the skin. As estrogen falls and swings through perimenopause, the band of body temperature the brain treats as "fine" narrows. Small rises in core temperature that it used to ignore now trip the heat-loss response: blood vessels in the face, neck and chest open wide, you sweat, and a few minutes later it passes.1

That is vasodilation, and on its own it is harmless. The colour is blood close to the surface, and it fades when the vessels close again.

What matters for skin is the repetition. In the Study of Women's Health Across the Nation, which followed more than 3,000 women through the transition, frequent hot flushes lasted a median of 7.4 years, and longest, more than 11 years, in women whose flushes began in perimenopause.2 Thousands of flushes, over years, on the same patch of skin.

2. Vessels that open often stay more reactive

Facial blood vessels that dilate many times a day, for years, become easier to trigger and slower to close. Dermatologists see this as the shift from flushing that comes and goes to redness that stays, often with fine visible vessels. Those are two of the defining features of rosacea in the current classification: fixed redness in the centre of the face that periodically intensifies, and flushing and visible vessels alongside it.3

The link to menopause is well observed, if not yet measured in a controlled trial. A 2022 review of menopause and skin disorders notes that research on menopause and rosacea is still thin, but that menopausal flushing is known to worsen rosacea and can even set it off in women who did not have it.4 Rosacea is an inflammatory condition, so for the women in whom it tips over, the redness does involve inflammation. The flush is the trigger; the inflammation is what the trigger, repeated, can produce.

3. Falling estrogen thins the barrier

The third piece has nothing to do with flushing at all. Estrogen acts directly on skin. Skin cells carry estrogen receptors, and facial skin carries more of them than skin on the body.5 As estrogen declines, skin thins, holds less water, and makes fewer of the lipids that seal the barrier.5

Stress hormones compound it. Cortisol reduces barrier lipids and hydration and increases water loss; in one study, students' skin barriers measurably took longer to recover during exam week than during holidays.6,7 A thinner barrier lets more through, so heat, wind, friction and products the skin used to ignore now register, and the response is redness.

Put together

"Red and flushed, most of the time" is usually these three stacked:

  • A barrier made thinner and more reactive by falling estrogen and rising cortisol, so ordinary things provoke colour.
  • Vessels trained open by years of flushing, so the colour lingers.
  • In some women, rosacea moving from occasional to constant.

The flush is a contributor and a trigger. It is not the whole story, which is why skin can stay red on days with no flush at all.

What this means for a routine

None of this changes hormones, and skincare does not either. What skin in this state needs is less to react to and more of what the barrier is losing. That means no added fragrance (the American Academy of Dermatology names fragrance allergy the leading cause of contact dermatitis from cosmetics8), ingredients that soothe the look of redness, such as bisabolol, panthenol, centella and aloe, support for the barrier from niacinamide and lightweight barrier lipids, and nothing that peels, strips or heats the skin.

That is how Cōpḍī's two formulas are built. Prep carries bisabolol, panthenol and aloe; Plump carries niacinamide and centella; both carry nigella and prickly pear botanicals, which replenish barrier lipids, and no added fragrance.

FAQ

Is a hot flush the same as inflammation?

No. A hot flush is vasodilation: blood vessels in the face, neck and chest open to release heat, and the colour fades when they close again.

Can menopause bring on rosacea?

Research is still thin, but a 2022 review notes that menopausal flushing is known to worsen rosacea and can even set it off in women who did not have it.

Why is my skin red even on days without a hot flush?

Falling estrogen and higher cortisol thin the barrier, so heat, wind, friction and products the skin used to ignore now provoke redness.

This article is for general information and isn't medical advice. For concerns about your skin or health, talk to your doctor or dermatologist.

Sources

  1. Freedman RR. Menopausal hot flashes: mechanisms, endocrinology, treatment. Journal of Steroid Biochemistry and Molecular Biology, 2014;142:115–120. PMID 24012626.
  2. Avis NE, Crawford SL, Greendale G et al. Duration of menopausal vasomotor symptoms over the menopause transition. JAMA Internal Medicine, 2015;175(4):531–539. PMID 25686030.
  3. Gallo RL, Granstein RD, Kang S et al. Standard classification and pathophysiology of rosacea: the 2017 update by the National Rosacea Society Expert Committee. Journal of the American Academy of Dermatology, 2018;78(1):148–155. PMID 29089180.
  4. Kamp E, Ashraf M, Musbahi E, DeGiovanni C. Menopause, skin and common dermatoses. Part 2: skin disorders. Clinical and Experimental Dermatology, 2022;47(12):2117–2122. PMID 35727900.
  5. Thornton MJ. Estrogens and aging skin. Dermato-Endocrinology, 2013;5(2):264–270. PMID 24194966.
  6. Garg A, Chren MM, Sands LP et al. Psychological stress perturbs epidermal permeability barrier homeostasis. Archives of Dermatology, 2001;137(1):53–59. PMID 11176661.
  7. Maarouf M, Maarouf CL, Yosipovitch G, Shi VY. The impact of stress on epidermal barrier function: an evidence-based review. British Journal of Dermatology, 2019;181(6):1129–1137. PMID 30614527.
  8. American Academy of Dermatology. Position statement on the chemical identity of fragrances (approved 1998). States that fragrance allergy is the number one cause of cosmetic contact dermatitis in the US.